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On January 29th, Liu Hongtao Of the Center for Excellence in Molecular Plant Sciences of the Chinese Academy of Sciences/Institute of Plant Physiology and Ecology, with UVR8 interacts with WRKY36 to regulate HY5 phirion and hypocotyl elongation in Arabidopsis, published online in Nature Plants, revealed a new mechanism for ultraviolet UV-B to regulate plant light patterns.
UV UVB can be used as a signal to regulate plant development, such as inhibiting plant elongation, promoting the accumulation of sub-leaf opening, flavonoids and anthocyanins.
UVR8 (UV RESISTANCE LOUCS 8) is a UV uv UVB receptor in plants and the last of all light receptors to be discovered, and was not reported until 2011, and the exact signaling mechanism is not clear.
UVR8 is activated by UV-B irradiation, changed from a dimmer to a monomer and enriched in the nucleus.
UVR8 must be in the nucleus to function, whether it directly regulates gene transcription in the nucleus needs further research.
Liu Hongtao's research team found that UVR8 can be directly combined with a new, functionally unknown transcription factor, WRKY36 (WRKY DNA-BINDING PROTEIN 36), in the form of non-dependent UV-B.
UV-B exposure promotes the formation of the UVR8-WRKY36 complex in the nucleus.
WRKY36 is essential for plant response UV-B, WRKY36's missing mutants under white light conditions the lower embryo shaft is shorter, but under UV-B conditions the lower embryo shaft elongation is basically consistent with wild type, indicating that WRKY36 is the positive regulatory factor of lower embryo shaft elongation, negative regulation UV-B inhibited lower embryo shaft elongation. Further research
showed that WRKY36 promotes growth by directly inhibiting the expression of the key gene HY5 in light morphology, uv-B-activated UVR8 forms monomers, and enters the nucleus directly binding WRKY36 and inhibits its binding HY5 promoter, thus promoting HY5 expression and inhibiting elongation.
these results show that WRKY36 is a negative regulatory factor for HY5 and light form construction, while UV-B directly inhibits WRKY36 through UVR8, thus promoting HY5 expression and light formation. The combination of
UVR8 and WRKY36 is an early mechanism for UV-B signal transduction, and UVR8-WRKY36-HY5 is a new UV-B signal path.
the above studies show that the direct binding of UVR8 to transcription factoris is the early mechanism of signal transduction, UVR8 can function in the nucleus because it is to directly bind the transcription factor WRKY36 and directly regulate transcription in the nucleus.
research work has been supported by the Ministry of Science and Technology, the National Natural Science Foundation of China and the Chinese Academy of Sciences.
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