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    Home > Food News > Food Articles > Chinese scholars have discovered the mechanism of brain damage caused by bisphenol A, the raw material of plastic products.

    Chinese scholars have discovered the mechanism of brain damage caused by bisphenol A, the raw material of plastic products.

    • Last Update: 2020-09-17
    • Source: Internet
    • Author: User
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    Original title: Chinese scholars have discovered the mechanism of brain damage caused by BPA, the raw material of plastic products
    BPA is a chemical raw material widely used in the production of food boxes, water pipes and other plastic products, which induces nervous system-related learning and memory disorders. Through experiments recently, the team of Associate Professor Hu Fans of Hefei University of Technology has discovered the neurophysiological mechanism of BPA interfering with brain function, which provides a theoretical basis and breakthrough for clinical evaluation and prevention. The results were published in advanced science and toxicology archives, international academic journals.
    as an environmental estrogen, BPA induces nervous system-related learning and memory disorders, but its physiological mechanisms have not been identified in academia for a long time. A recent experiment by Associate Professor Hu Fans of the School of Food Science and Engineering, Hefei University of Technology, found that young rats between 21 and 49 days after birth continued to be exposed to bisphenol A at a dose of 1 mg per kilogram of body weight per day, and were responsible for learning and remembering the synapses of cone neurons in the haima region. Density decreased by 28.8%, pre-synapse release and post-synapse subject expression decreased by 16.4%, long-range synactic gain decreased by 33.8%, memory behavior performance decreased by 17.4%, resulting in severe spatial memory impairment and physiological function changes.
    same time, they worked with the Visual Research Laboratory at the Chinese University of Science and Technology to find that cats were weighing 0 per kg. After 2 hours of exposure to BPA at a dose of 4 mg, there was a significant increase in the level of knee projection and in-cortical inhibition in the lower cerebral cortical cortical layer, resulting in a decrease in the adiroid selectivity of the primary visual cortical layer and the fidelity of signal transmission.
    , the experimental use of young rats equivalent to the human 1 to 12 years of age adult growth, and the use of BPA dose is lower than the U.S. Food and Drug Administration no systemic toxicity dose standard.
    the results suggest that the brain has been damaged at the source of information input by BPA interference. By revealing the neurophysiological mechanism of BPA interfering with brain function, the results provide a theoretical basis and breakthrough for clinical evaluation and prevention of brain function damage caused by BPA. (Xu Haitao)
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