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recently, the Chinese Academy of Sciences Center for Excellence in Molecular Plant Science Innovation Center / Institute of Plant Physiology and Ecology Liyuan research team revealed LTF1 as a litopic synthesis induction switch to regulate the molecular mechanism of ligand synthesis. The findings were published online in Molecular Plants.
ligand synthesis is specifically activated in the cells that form secondary cell walls, providing mechanical support for the upright growth of plants, helping to establish long-distance transport routes of water and substances, and strengthening the plant's resistance barrier. In thin-walled cells without secondary cell walls, the glucosin synthesis pathway is closed. How does the italic synthesis pathway start and close? This is an important issue that has been widely concerned but has not been resolved.
researchers found that a MYB transcription factor, LTF1, binds directly to the initiator of a ligand synthesis gene, inhibiting ligand synthesis. "LTF1 can be activated by MPK6 phosphorylation, and phosphorylation LTF1 degrades through protease pathways, removing inhibition of ligand synthesis gene expression and initiating ligand synthesis pathways." The researchers explained that a variety of internal and external signals can activate MPK6, LTF1 through MPK6 to sense a variety of signals, regulating the closure and activation of the anabolic pathway of the herb.
this discovery provides a new operational technical pathway for the precise and directional modification of cell walls, which can be used to cultivate new varieties of trees and crops with ideal properties.