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Differential diagnosis of T1 hyperintensity liver disease

 Last Update: 2021-11-01
 Source: Internet
 Author: User

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Differential diagnosis : T1 hyperintensity liver disease

Differential diagnosis : T1 high signal liver disease diagnosis

Differential Diagnosis: T1 Bright Liver Lesions Differential Diagnosis: T1 Bright Liver Lesions

①

① ①

T1 high signal with black ring, T1 high signal with black ring, no enhancement

T1 bright with a dark rim
T2 bright with a dark rim
No enhancement
No enhancement

T1 bright with a dark rim

T2 bright with a dark rim

No enhancement

Postoperative hematoma

Postoperative hematomahematoma

Hematoma after surgery

Hematoma after surgery Hematoma after surgery

②

② ②

T1 high signal and low signal focus, T2 low signal and good signal focus, heterogeneous enhancement of the focus, clear solid part

T1 very bright within a dark lesion
T2 dark with a bright lesion
Heterogeneous enhancement within the lesion
Washout within the solid lesion

T1 very bright within a dark lesion

T2 dark with a bright lesion

Heterogeneous enhancement within the lesion

Washout within the solid lesion

Carcinoid liver metastases with bleeding

Carcinoid liver metastases with bleeding Carcinoid liver metastases with bleeding

Hemorrhagic carcinoid metastasis

Hemorrhagic carcinoid metastasis Hemorrhagic carcinoid metastasis

③

③ ③

T1 high signal and low signal focus, T2 mixed signal, irregular ring enhancement, heterogeneous, continuous enhancement

T1 bright and dark lesions
T2 mixed signal intensity
Irregular ring-shaped enhancement
Heterogeneous and persistent enhancement

T1 bright and dark lesions

T2 mixed signal intensity

Irregular ring-shaped enhancement

Heterogeneous and persistent enhancement

Carcinoid liver metastases and protein production

Protein-producing carcinoid metastasis Protein-producing carcinoid metastasis

④

④ ④

T1 is mainly high signal, T2 waits to bright high signal, T1 high signal part is strengthened, heterogeneity continues to strengthen

T1 (predominantly) bright lesions
T2 isointense to very bright
Enhancement of the T1-bright parts
Heterogeneous and persistent enhancement

T1 (predominantly) bright lesions

T2 isointense to very bright

Enhancement of the T1-bright parts

Heterogeneous and persistent enhancement

Melanoma liver metastasis

Melanoma liver metastases Melanoma liver metastases

Melanoma metastases

Melanoma metastases Melanoma metastases

summary

Summary summary

Evaluation T1WI: All lesions have high signal components in the non-cirrhotic liver
Evaluation on T2WI: In the first case, the lesion was surrounded by a low-signal edge with increased thickness
Assessing the enhanced arterial phase: the first case did not have any enhancement, the second case showed uneven enhancement, and the solid part around the lesion was thicker in the third and fourth cases with enhancement
Assess the delayed enhancement period: the first case showed no enhancement, the second case was clear with continuous uneven enhancement, and the third and fourth cases continued to have uneven enhancement at the edge of the lesion

Evaluation T1WI: All lesions have high signal components in the non-cirrhotic liver

Evaluation on T2WI: In the first case, the lesion was surrounded by a low-signal edge with increased thickness

Assessing the enhanced arterial phase: the first case did not have any enhancement, the second case showed uneven enhancement, and the solid part around the lesion was thicker in the third and fourth cases with enhancement

Assess the delayed enhancement period: the first case showed no enhancement, the second case was clear with continuous uneven enhancement, and the third and fourth cases continued to have uneven enhancement at the edge of the lesion

Hematoma: T1 high signal is due to methaemoglobin, and there is a hemosiderin ring at the edge;
Hemorrhagic carcinoid metastasis, protein-producing carcinoid metastasis: suggesting metastasis; no specific manifestations; combined with clinical
.
Melanoma metastasis: combined with medical history, T1 high signal; typical arterial phase is obviously enhanced
.

Hematoma: T1 high signal is due to methaemoglobin, and there is a hemosiderin ring at the edge;

Hemorrhagic carcinoid metastasis, protein-producing carcinoid metastasis: suggesting metastasis; no specific manifestations; combined with clinical
.

Melanoma metastasis: combined with medical history, T1 high signal; typical arterial phase is obviously enhanced
.

In addition, hepatocellular adenomas are prone to hemorrhage, and can also be manifested as T1 hyperintensity, clear borders, obvious enhancement in the arterial phase, iso-signal in the delayed phase, and surrounding pseudo-capsules
.
When combined with massive bleeding, the residual solid part is strengthened
.

In addition, hepatocellular adenomas are prone to hemorrhage, and can also be manifested as T1 hyperintensity, clear boundaries, obvious enhancement in the arterial phase, and iso-signal in the delayed phase, and the surrounding pseudocapsules are seen
.
When combined with massive bleeding, the residual solid part is strengthened
.

☆ Content source: English book "Liver MRI" ☆ Content source: English book "Liver MRI"

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