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*Only for medical professionals to read and reference today's article is "hot pot flavor".
.
.
Winter is here, and hot pot has become the first choice for many families or friends to have dinner together.
Although hot pot is delicious, it is good for patients with high uric acid.
Unable to reach the dust"-After eating a hot pot, the purine should be "choky" and go up, and then gout is inevitable! Don't panic! Today’s article is here to chat with everyone, "How should friends with high uric acid choose hot pot" and "What should I do if I accidentally eat high uric acid"! Knowing where the purine comes from, you will know how to choose hot pot! Take several common pot bottoms on the market as an example: As the bottom of the clear soup pot contains only white water, green onions, ginger, wolfberry, etc.
, the purine content of the soup itself (calculated at 1 mg/100 g) is in single digits, which is low enough to be negligible.
The degree is most suitable for people who need to control uric acid
.
Tomato sauce is added to the bottom of the tomato soup pot, which is a low-purine ingredient, so it does not affect the purine content of the soup
.
The large amount of potassium in tomatoes is also very conducive to the smooth excretion of uric acid from the body
.
But in addition to tomatoes, the umami seasonings added are worthy of attention
.
Because chicken essence, yeast hydrolysate and other freshness enhancers contain umami nucleotides, and the nucleotides must contain purines
.
Therefore, its purine content will be higher than that of clear soup, but it is still acceptable
.
Where is the bottom of the broth? Everyone knows that fungi are high in purine content in plant foods
.
According to the measurement data in China, the purine content per 100 grams of fresh fungus is between 21-79 mg, and the content of dry fungus is between 68-569 mg because of the concentration of "dry goods"
.
A soup boiled with a lot of fresh and dried bacteria will contain a lot of purines
.
Although there is no measurement data of the mushroom soup, it is much higher than the clear soup.
The specific content depends on the type of mushroom, the specific amount and the boiling concentration
.
In general, the bottom of the mushroom soup pot is not suitable for diners who need to control uric acid
.
Fish soup, broth, chicken broth, and bone broth are completely unsuitable for those who control uric acid, because during the boiling process, the purines in fish, meat, and chicken will gradually dissolve into the soup, and there may be many additions to it.
Add fresh seasonings, so this kind of pot bottom tastes richer and more delicious, and the purine content is more worrying
.
Butter soup and clear oil soup base are not suitable for those who need to control uric acid
.
Butter and clear oil are fats, and purines are insoluble in fats, so the purine content of the oil itself is very low
.
However, a small amount of broth and a variety of seasonings are usually added to the bottom of the oil soup pot
.
In addition, most people with high uric acid are also accompanied by overweight and obesity, fatty liver, and hyperlipidemia, and a large amount of saturated fat is not conducive to blood lipid control
.
Why does uric acid keep increasing when diet is absent? Then there are patients who want to ask, I neither like to eat hot pot, and I have a good diet control, why does my uric acid rise instead of falling? This is because about 80% of blood uric acid in the human body comes from self-synthesis, and 20% comes from foreign food
.
Therefore, the effect of lowering uric acid by only controlling diet is still relatively limited
.
Uric acid levels are affected by many factors such as age, gender, race, genetics, eating habits, drugs, environment, etc.
Diet only accounts for part of the source of uric acid, so high-purine diets such as hot pot, seafood, and beer are just one factor that causes hyperuricemia The decrease in uric acid excretion is the common cause of hyperuricemia
.
As age increases, various diseases such as high blood pressure and diabetes can cause a decrease in glomerular filtration rate and a decrease in the secretion of uric acid in the renal tubules, leading to uric acid excretion disorders
.
Therefore, in addition to a low-purine diet, you should also focus on weight control, avoid alcoholism, overwork, trauma, cold, and mental stress.
Drinking more water and regular exercise can reduce uric acid
.
If blood uric acid is too high or some complications such as gout, you need to take drugs such as allopurinol to control uric acid
.
How to choose drugs for gout patients? At present, western medicine treats gout mainly from three aspects: pain relief, lowering uric acid, and alkalizing the internal environment
.
For patients diagnosed with gout, regardless of whether the uric acid exceeds the standard, they need uric acid lowering treatment.
Only when the uric acid is lowered can patients hope to cure gout.
A reasonable level of uric acid will not cause damage to the kidneys, cardiovascular and cerebrovascular organs, and reduce gout.
The probability of seizures also avoids the complications of hyperuricemia
.
Analgesic and gout are mainly used to relieve inflammation and relieve symptoms in the acute stage
.
Non-steroidal anti-inflammatory analgesics are the first drugs recommended in the guidelines to relieve symptoms.
Commonly used are diclofenac sodium, ibuprofen, indomethacin, meloxicam, celecoxib, etc.
This class of drugs is mainly suitable for gout patients whose pain symptoms are not particularly severe
.
When the effect of non-steroidal anti-inflammatory drugs is not ideal, some doctors will choose the more potent colchicine.
If the effect is not satisfactory or there are contraindications to medication, they may need to choose glucocorticoids
.
Such drugs include hydrocortisone (short-acting class), prednisolone (medium-acting class), and dexamethasone (long-acting class).
Combination with non-steroidal anti-inflammatory and analgesics is not recommended
.
Once gout has an acute attack, patients should use anti-inflammatory and analgesic drugs within 24 hours
.
Studies have shown that taking colchicine more than 36 hours is less effective
.
European gout diagnosis and treatment guidelines believe that it is best to start drug treatment within 12 hours after the onset, and it is strongly recommended that gout patients carry drugs for the treatment of gout attacks
.
Lowering uric acid and lowering blood uric acid levels are the key to controlling gout attacks.
Increasing uric acid excretion and inhibiting uric acid production are two commonly used methods
.
When gout patients undergo uric acid-lowering treatment, drugs that inhibit uric acid production are recommended to use allopurinol or febuxostat; drugs that promote uric acid excretion are recommended to use benzbromarone
.
And doctors should use the above-mentioned uric acid-lowering drugs in a targeted manner according to the specific conditions of the patient, and be alert to possible liver and nephrotoxicity and other side effects during the medication
.
(The selection strategy is shown in Figure 1) Figure 1 For gout patients with chronic kidney disease, it is recommended to evaluate renal function first, and then use uric acid-lowering drugs that have little effect on renal function according to the specific conditions of the patient, and closely monitor adverse reactions during treatment
.
Drugs that inhibit uric acid production (allopurinol and febuxostat) and drugs that promote uric acid excretion (benzbromarone) can reduce glomerular uric acid load
.
When allopurinol is used in patients with renal insufficiency, the starting dose should be reduced, and the dose should be gradually increased, and closely monitored for hypersensitivity reactions
.
When febuxostat is applied to patients with mild to moderate renal insufficiency, there is no need to adjust the dose
.
Uric acid excretion drugs should be used with caution in patients with uric acid kidney stones and patients with severe renal insufficiency
.
Alkalizing urine "Chinese Guidelines for the Diagnosis and Treatment of Hyperuricemia and Gout (2019)" pointed out that when the morning urine pH value of patients with hyperuricemia and gout is less than 6.
0, it is recommended to take citric acid preparations and sodium bicarbonate to alkalize urine To maintain the morning urine pH at 6.
2-6.
9 to reduce the risk of uric acid kidney stones and facilitate the dissolution of uric acid kidney stones
.
Its usage is as follows: References: [1] "2016 Guidelines for the Diagnosis and Treatment of Gout in China" [2] Guidelines for the Diagnosis and Treatment of Hyperuricemia and Gout in China (2019)
.
.
Winter is here, and hot pot has become the first choice for many families or friends to have dinner together.
Although hot pot is delicious, it is good for patients with high uric acid.
Unable to reach the dust"-After eating a hot pot, the purine should be "choky" and go up, and then gout is inevitable! Don't panic! Today’s article is here to chat with everyone, "How should friends with high uric acid choose hot pot" and "What should I do if I accidentally eat high uric acid"! Knowing where the purine comes from, you will know how to choose hot pot! Take several common pot bottoms on the market as an example: As the bottom of the clear soup pot contains only white water, green onions, ginger, wolfberry, etc.
, the purine content of the soup itself (calculated at 1 mg/100 g) is in single digits, which is low enough to be negligible.
The degree is most suitable for people who need to control uric acid
.
Tomato sauce is added to the bottom of the tomato soup pot, which is a low-purine ingredient, so it does not affect the purine content of the soup
.
The large amount of potassium in tomatoes is also very conducive to the smooth excretion of uric acid from the body
.
But in addition to tomatoes, the umami seasonings added are worthy of attention
.
Because chicken essence, yeast hydrolysate and other freshness enhancers contain umami nucleotides, and the nucleotides must contain purines
.
Therefore, its purine content will be higher than that of clear soup, but it is still acceptable
.
Where is the bottom of the broth? Everyone knows that fungi are high in purine content in plant foods
.
According to the measurement data in China, the purine content per 100 grams of fresh fungus is between 21-79 mg, and the content of dry fungus is between 68-569 mg because of the concentration of "dry goods"
.
A soup boiled with a lot of fresh and dried bacteria will contain a lot of purines
.
Although there is no measurement data of the mushroom soup, it is much higher than the clear soup.
The specific content depends on the type of mushroom, the specific amount and the boiling concentration
.
In general, the bottom of the mushroom soup pot is not suitable for diners who need to control uric acid
.
Fish soup, broth, chicken broth, and bone broth are completely unsuitable for those who control uric acid, because during the boiling process, the purines in fish, meat, and chicken will gradually dissolve into the soup, and there may be many additions to it.
Add fresh seasonings, so this kind of pot bottom tastes richer and more delicious, and the purine content is more worrying
.
Butter soup and clear oil soup base are not suitable for those who need to control uric acid
.
Butter and clear oil are fats, and purines are insoluble in fats, so the purine content of the oil itself is very low
.
However, a small amount of broth and a variety of seasonings are usually added to the bottom of the oil soup pot
.
In addition, most people with high uric acid are also accompanied by overweight and obesity, fatty liver, and hyperlipidemia, and a large amount of saturated fat is not conducive to blood lipid control
.
Why does uric acid keep increasing when diet is absent? Then there are patients who want to ask, I neither like to eat hot pot, and I have a good diet control, why does my uric acid rise instead of falling? This is because about 80% of blood uric acid in the human body comes from self-synthesis, and 20% comes from foreign food
.
Therefore, the effect of lowering uric acid by only controlling diet is still relatively limited
.
Uric acid levels are affected by many factors such as age, gender, race, genetics, eating habits, drugs, environment, etc.
Diet only accounts for part of the source of uric acid, so high-purine diets such as hot pot, seafood, and beer are just one factor that causes hyperuricemia The decrease in uric acid excretion is the common cause of hyperuricemia
.
As age increases, various diseases such as high blood pressure and diabetes can cause a decrease in glomerular filtration rate and a decrease in the secretion of uric acid in the renal tubules, leading to uric acid excretion disorders
.
Therefore, in addition to a low-purine diet, you should also focus on weight control, avoid alcoholism, overwork, trauma, cold, and mental stress.
Drinking more water and regular exercise can reduce uric acid
.
If blood uric acid is too high or some complications such as gout, you need to take drugs such as allopurinol to control uric acid
.
How to choose drugs for gout patients? At present, western medicine treats gout mainly from three aspects: pain relief, lowering uric acid, and alkalizing the internal environment
.
For patients diagnosed with gout, regardless of whether the uric acid exceeds the standard, they need uric acid lowering treatment.
Only when the uric acid is lowered can patients hope to cure gout.
A reasonable level of uric acid will not cause damage to the kidneys, cardiovascular and cerebrovascular organs, and reduce gout.
The probability of seizures also avoids the complications of hyperuricemia
.
Analgesic and gout are mainly used to relieve inflammation and relieve symptoms in the acute stage
.
Non-steroidal anti-inflammatory analgesics are the first drugs recommended in the guidelines to relieve symptoms.
Commonly used are diclofenac sodium, ibuprofen, indomethacin, meloxicam, celecoxib, etc.
This class of drugs is mainly suitable for gout patients whose pain symptoms are not particularly severe
.
When the effect of non-steroidal anti-inflammatory drugs is not ideal, some doctors will choose the more potent colchicine.
If the effect is not satisfactory or there are contraindications to medication, they may need to choose glucocorticoids
.
Such drugs include hydrocortisone (short-acting class), prednisolone (medium-acting class), and dexamethasone (long-acting class).
Combination with non-steroidal anti-inflammatory and analgesics is not recommended
.
Once gout has an acute attack, patients should use anti-inflammatory and analgesic drugs within 24 hours
.
Studies have shown that taking colchicine more than 36 hours is less effective
.
European gout diagnosis and treatment guidelines believe that it is best to start drug treatment within 12 hours after the onset, and it is strongly recommended that gout patients carry drugs for the treatment of gout attacks
.
Lowering uric acid and lowering blood uric acid levels are the key to controlling gout attacks.
Increasing uric acid excretion and inhibiting uric acid production are two commonly used methods
.
When gout patients undergo uric acid-lowering treatment, drugs that inhibit uric acid production are recommended to use allopurinol or febuxostat; drugs that promote uric acid excretion are recommended to use benzbromarone
.
And doctors should use the above-mentioned uric acid-lowering drugs in a targeted manner according to the specific conditions of the patient, and be alert to possible liver and nephrotoxicity and other side effects during the medication
.
(The selection strategy is shown in Figure 1) Figure 1 For gout patients with chronic kidney disease, it is recommended to evaluate renal function first, and then use uric acid-lowering drugs that have little effect on renal function according to the specific conditions of the patient, and closely monitor adverse reactions during treatment
.
Drugs that inhibit uric acid production (allopurinol and febuxostat) and drugs that promote uric acid excretion (benzbromarone) can reduce glomerular uric acid load
.
When allopurinol is used in patients with renal insufficiency, the starting dose should be reduced, and the dose should be gradually increased, and closely monitored for hypersensitivity reactions
.
When febuxostat is applied to patients with mild to moderate renal insufficiency, there is no need to adjust the dose
.
Uric acid excretion drugs should be used with caution in patients with uric acid kidney stones and patients with severe renal insufficiency
.
Alkalizing urine "Chinese Guidelines for the Diagnosis and Treatment of Hyperuricemia and Gout (2019)" pointed out that when the morning urine pH value of patients with hyperuricemia and gout is less than 6.
0, it is recommended to take citric acid preparations and sodium bicarbonate to alkalize urine To maintain the morning urine pH at 6.
2-6.
9 to reduce the risk of uric acid kidney stones and facilitate the dissolution of uric acid kidney stones
.
Its usage is as follows: References: [1] "2016 Guidelines for the Diagnosis and Treatment of Gout in China" [2] Guidelines for the Diagnosis and Treatment of Hyperuricemia and Gout in China (2019)
