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So how did these viruses come to life and be implanted into the baby's intestines? Will delivery and feeding have an impact on the disease of the virus?recently, researchers from the University of Pennsylvania recently conducted a study on the origin and geriate of early neonatal enteroviruses, and found that early implantation of infant enteroviruses is gradual, the first stage induces the production of early bacterial phages, the second stage of the presence of human cells infected with the virus, which can be breastfed to inhibit the accumulation of enterovirus, reduce the occurrence of enterovirus infection and viral gastroenteritisThe study was published in Nature on April 15, local timeTo study early viral implantation, the researchers used fluorescent staining to analyze stool samples from 20 healthy African-American babies during the three-year period after birthIt was found that viral-like particles (VLP) were not basically detected in early infant feces, but VLP reached 1.6 x 109 per 10g of feces at 1 month of age, and VLP was similar to that of adults in the gut, which may persist throughout lifethen the researchers used qPCR and macro genome sequencing to analyze the microbiome of virus-rich samples to study the origin of virus populationsIt was found that bacterial richness and bacterial diversity had a strong positive correlation with VLP count, virus richness and virus diversityEarly in life, pioneering bacteria (including Bacteria, Linebacteria, Tocoytites, and thick wall bacteria) are planted in the baby's intestines and then induce mild phages to develop gradually, becoming the main population in the intestines a month laterBy the fourth month, viruses (including herpes viruses, adenoviruses, ring viruses, cupviruses, and small RNA viruses) are gradually emergingdetection and characteristics of VLP in infant intestinal stool samples
and then, the researchers quantitatively and genomeally sequenced VLP produced by the culture of 24 strains of infant feces, and found that bacteria in the infant's intestines were able to produce a higher level of VLP after phage induction, and that the virus group was consistent with the detection in the fecal samples, indicating that the virus was induced by phagephage induced early-stage virus
the researchers also compared the characteristics of VLP data in 125 infant feces with feeding history, delivery patterns, and so onIt was found that there was no significant effect on the mode of delivery, and that only 9% of infants with breast milk or mixed milk and formula fed tested positive for the virus in 30% of formula-fed infants, and an increase in mild phages of probiotic scophycoccal bifico and Lactobacillus, suggesting that breastfeeding inhibited viral growth and accumulation and protected intestinal healthbreastfeeding and infant enterovirusin short, neonatal enterovirus is developed in the early stages, and immunofactor-rich breastfeeding can inhibit the infant's intestinal virus planting, the results also provide a new strategy for the prevention of early gastrointestinal diseasesreference: si aaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaaadh snr Original title: Nature: Can breastfeeding regulate neonatal enterovirus implantation?