Prosthyroids, thrombosis and white triene.

name
prostaglandin (prostaglandin, PG), thromboxane (TX) and leukotrienes (LT) are derived from peanut tetrylic acid. When they are produced in cells, they act as regulators to regulate almost all cell metabolism and are associated with pathological processes such as inflammation, allergic reactions, and cardiovascular disease.film on the biofilm contains peanut tyrenic acid, which can be hydrolyzed by phospholipidase A2, releasing peanut tyrenic acid. Peanut tyrenic acid can be catalyzed by peroxidation synthase in prostatin, consuming O2 and reductive glutathione, oxidation and cyclification reactions occur, resulting in prostatin H2. Prosupaline H2 can be further derived from other prosthyroids and thrombosis.
cortisol inhibits the activity of
phosphatase
A2, reducing the production of peanut tetraoleic acid, thereby inhibiting the synthesis of prostatin, aspirin and phenylbutazone inhibiting the activity of peroxidation synthase in prostatin to reduce prostatin and thrombosis.. Physiological functions of PG, TX and LTPG are very low in cells, only 10pmol/L, but have strong physiological activity.(i) PG PGE2 can induce inflammation, promote local vascular dilation, capillaries permeability increased, causing redness, swelling, pain, heat and other symptoms. PGE2 and PGA2 make the arteries smooth muscle throes, which have the effect of lowering blood pressure, and PGE2 and PGI2 inhibit the secretion of stomach acid and promote smooth muscle peristalsis in the gastrointestinal intestinal intestinal intestinal.PGE2 and PGE2 alpha produced by
follicles play an important role in the ovulation process, PGE2 alpha can smooth muscle contraction of the ovaries, cause ovulation, the release of PGE2 alpha of the uterus can dissolve the yellow body, the endometrium release of PGE2 alpha during childbirth can cause the uterine contraction to strengthen, promote childbirth.(ii) TXplateplate production of TXA2 and PGE2 promote plateplate aggregation, blood vessel contraction, promote clotting and thrombosis, while PGI2 released by vascular endotrkin cells has a strong vascular and antiplateplate plateaggregation, inhibit clotting and thrombosis, and the role of TXA2.
Arctic Eskimos eat blood foods rich in peanut tyrenic acid, which can be synthesized in the body PGE3, PGI3 and TXA3 and other three
compounds
. PGI3 inhibits the release of peanut trenoleic acid from membrane phospholipids, thus inhibiting the synthesis of PGI2 and TXA2. Since PGI3 has the same activity as PGI2, and TXA3 is much weaker than TXA2, Eskimos have strong antiplatelet aggregation and anticoagulant effect, which is considered to be one of the important reasons why they are less prone to myocardial infarction.(iii) LThas been shown to be a mixture of LTC4, TD4 and LTE4, which makes the bronchial smooth muscle contraction 100,000 times stronger than histamine and PGF2, and the effect is slow and long-lasting. In addition, LTG4 can also regulate the function of white blood cells, promote their wandering and metastatic action, stimulate adenosine cyclase, induce multi-nucleocyte departitation, so that lysase release hydrolyzed enzymes, promote the development of inflammation and allergic reactions.
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