-
Categories
-
Pharmaceutical Intermediates
-
Active Pharmaceutical Ingredients
-
Food Additives
- Industrial Coatings
- Agrochemicals
- Dyes and Pigments
- Surfactant
- Flavors and Fragrances
- Chemical Reagents
- Catalyst and Auxiliary
- Natural Products
- Inorganic Chemistry
-
Organic Chemistry
-
Biochemical Engineering
- Analytical Chemistry
- Cosmetic Ingredient
-
Pharmaceutical Intermediates
Promotion
ECHEMI Mall
Wholesale
Weekly Price
Exhibition
News
-
Trade Service
Western high-fat diets are often associated with heart vascular diseases, One explanation is that the members of the gut microbiota dietary choline is decomposed into trimethylamine (TMA), trimethylamine absorbed in the intestine, in the liver It is oxidized to trimethylamine N-oxide (TMAO), which is a metabolite that promotes arteriosclerosis
Heart vascular trimethylamine absorbed in the intestine, is oxidized to N- trimethylamine-oxide (of TMAO) in the liver, promote atherosclerosis which is a metabolite of trimethylamine are absorbed in the intestine, it is oxidized in the liver Trimethylamine N-oxide (TMAO), which is a metabolite that promotes arteriosclerosis
Currently, a key but unexplored aspect of the TMAO pathway is how the interaction between host physiology and microbial communities due to impaired diet affects the production of TMA
In addition to changing the composition of the microbiota, a high-fat diet also changes the physiological condition of the host, because saturated fatty acids can damage the energy production of the mitochondria by inducing the mitochondria to produce hydrogen peroxide
Saturated fatty acids can damage the energy production of mitochondria by inducing mitochondria to produce hydrogen peroxide.
A high-fat diet changes the physiological structure of epithelial cells and increases the availability of electron receptors in the host's respiratory cavity
A high-fat diet changes the physiological structure of epithelial cells and increases the availability of the host's respiratory electron receptors.Long-term exposure to a high-fat diet can change the physiological structure of the intestinal epithelium and upgrade the choline decomposition effect of E.
The choline decomposition of E.
A high-fat diet can cause colon cell dysfunction and make the microbiota produce more trimethylamine N-oxide, which is a potential factor that causes cardiovascular disease in individuals
Original source:
Original source:WOONGJAE YOO et al.
WOONGJAE YOO et al.
Leave a message here