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Picture source: Reference [1])
On the other hand, mice lacking MARK4 can maintain a considerable percentage of left ventricular ejection fraction, indicating that their cardiac function is limited.
So is MARK4 related to the detyrosine effect of microtubules? In mice lacking MARK4, the researchers extracted samples with significantly lower levels of des-tyrosine microtubules.
▲This study is expected to find potential new targets for improving heart function after myocardial infarction (picture source: reference [1])
Finally, the researchers found the mechanism of MARK4.
Reference materials:
[1] Yu, X.