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Wild rape yellow monocytobacteria (Xcc) can cause the black rot of a variety of cruciferous plants such as amoeba, kale, and is an important model bacteria to study the molecular mechanism of plant interaction with pathogenic bacteria.
the Xcc genome has an xccR/pip genetic site, which is necessary for bacteria to perform pathogenic functions.
XccR is the homologous protein of the bacterial population-sensing transcription factor LuxR, known as the LuxR solo protein due to the absence of the corresponding LuxI signaling molecular syntase.
in the previous study, a research team led by Jia Yantao, a researcher at the Institute of Microbiology of the Chinese Academy of Sciences, under the guidance of Fang Rongxiang, a member of the Chinese Academy of Sciences, found that the absence of the pip gene (encoded proline carousilline peptidease) led to a decline in Xcc pathogenic force, and the expression of pip was induced in plants, and found that XccR by sensing plant signals, and in combination with the pip promoter's upstream Xluxbox, which is the first international study of the expression of the plant's trans-specy molecules.
new study found that: group-sensing LuxR solo protein was first found to directly regulate the expression of type III secretion system effect factor PIP, PIP in the process of regulating bacterial gene expression and plant host immunity has a dual role, that is, by coordinating their own internal and external environment, to "inside and outside" strategy to facilitate the complex mechanism of infecting plants: on the one hand, PIP as a negative bacterial motion negative Regulatory factors promote the accumulation of c-di-GMP, the second messenger in bacterial cells, and inhibit the expression of bacterial whiplash-related genes, on the other hand, when Xcc comes into contact with plant host cells, PIP protein simply acts as an effect factor to inject into plant cells through the type III secretion system, interfering with the disease-resistant signaling pathways of plant salicylic acid.
in the process of plant-pathogen interaction, bacteria reduce energy consumption by controlling their motority, and inject ingesting effect proteins into host cells with the most powerful weapon of bacteria, type III secretion system, to interfere with the immune response of plants, revealing the new mechanism of PIP to cope with plant environmental changes in the process of resistance to plant immunity. On March 8,
, the results were published online in Molecular Plant Pathology, based on the a-dual for proline for-proline iminopeptidase in the regulating-public-level and host immunity.
Fang Rongxiang's doctoral students Yan Jinhong and An Lin are the co-first authors of the paper, and Jia Yantao and Fang Rongxiang are the co-authors of the paper.
research work has been supported by the National Key Basic Research and Development Program (973 Program) and the National Natural Science Foundation.
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