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Biotechnology Channel News: Smoke is a widely present conditional pathogenic fungus in the natural environment, the resulting air-transmitting robes, can be sucked into the end of the respiratory tract, causing tritomy allergenic diseases, such as allergic asthma, allergic pneumonia, allergic bronchop-ulmonary aspergillosis, ABPA and other diseases.
At the same time, the increased concentration of icing mold spores in the environment is closely related to the exacerbation of disease symptoms, such as high concentrations of icing mold robes and asthma symptoms and increased death rate, in vivo experiments have been shown to cause asthma model rat airway high response and airway inflammation.
Fat cells are important effects and regulatory immune cells, which can release inflammatory media such as histamine, synthesize and release cytokines and trending factors, and are closely related to allergic diseases such as allergic asthma and measles.
currently known to contain 23 antigen components, can induce the body to produce specific IgE and combine with it on the surface of fat cells on the IgE receptomy, induce fat cells to departicle.
In recent years, studies have found that fat cells can also participate in the immunomodification of direct bacteria through a variety of non-IgE dependence active pathways, and fungi such as Candococ and Schenk robe silk bacteria have been found to live fertilizer cells through non-IgE dependence pathways.
causes fat cells to departitalize or synthesize and release related cytokines.
in-body experiments found that icing mycelium can directly induce hypertropic cells to de-pull, suggesting that in addition to the classic IgE pathway, there are non-IgE-dependent active pathways involved in the immunomodulating process of icing mold sensitivity.
this experiment, by studying the active effect of heating inactivated icing spore antigens on fat cells, increased the understanding of the immune regulation function of hypertropic cells during the allergy of icing.
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