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defects don't usually mean good things, but a new study in the United States has found that yeast cells live longer if there is some kind of "chromatin structural defect." The findings could help explore similar mechanisms in human cells and find anti-aging methods.
chromatin is present in the nucleocarbons and is the structure of DNA and histones. In the past, it has been suggested that low histone levels can easily lead to the dispersal of chromosomal structures, which is not conducive to cell longevity.
Researchers at Baylor College of Medicine in the U.S. recently published a paper in the American journal Science Advances that they used genetic techniques to remove certain histone genes from brewing yeast, causing a decline in the number of histones in cells, leading to some chromatin structural defects. The researchers thought yeasts with chromatin structural defects would live longer, but didn't expect them to live longer.
researchers also experimented with beautiful crypto-rod worms, and also found that this chromatin structural defect leads to longevity. The researchers believe this is because specific chromosomal structural defects activate a range of anti-aging genes that ultimately lead to longevity.
, an introviral anti-aging mechanism that may still exist in other organisms, told Xinhua that the paper's author and an assistant professor at Baylor College of Medicine, Shi Qi, told Xinhua. If similar mechanisms are found in human cells in the future, they are expected to be used to develop anti-aging methods. (
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