Alzheimer's & Dementia: The relationship between urban environmental exposure and Alzheimer's disease risk: a study of the population of Barcelona 

People living in urban areas are often exposed to high levels of air pollutants and noise, and lack contact with the natural outdoor environment
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Long-term exposure to air pollution and noise, and lack of green space are related to premature all-cause mortality and other harmful health effects

As we all know, air pollution is an important environmental hazard and a major environmental health problem
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Epidemiological studies have shown that exposure to ambient air pollution has an impact on the nervous system, and may cause causality through harmful effects on the brain, accelerating cognitive aging

Exposure to the urban environment may also contribute to the incidence of Alzheimer's disease (AD)
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Recently, researchers conducted a study based on the Barcelona population to determine the impact of urban environmental exposure on structural brain imaging among cognitively accessible people, and whether it increases the risk of AD

The study included 212 participants living in Barcelona, ​​Spain, and performed brain scans
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Researchers used land-use regression models to estimate residents’ exposure to air pollutants

The effect of exposure to air pollutants, noise, and proximity to green spaces on the volume of gray matter and white matter

The effect of exposure to air pollutants, noise, and proximity to green space on the volume of gray matter and white matter The effect of exposure to air pollutants, noise, and proximity to green space on the volume of gray and white matter

The results of the study showed that exposure to nitrogen dioxide is related to the lower GM volume in the pre-wedge zone and the larger WM volume in the corpus callosum and lower longitudinal tendons
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In contrast, exposure to fine particulate matter is associated with larger cerebellar GM and WM in the corpus callosum, superior longitudinal fasciculus, and cingulate gyrus

Exposure to nitrogen dioxide is associated with the lower GM volume in the pre-wedge zone and the larger WM volume of the corpus callosum and lower longitudinal ribs.

 

Original source:

Original source:

Carles Falcón et al.

Carles Falcón et al.

 

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