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18 years old, male
.
Quadriplegia below C6 after traumatic accident
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2 sudden 30min thunderclap headaches, vision loss, new onset urinary incontinence, hypertension (200s/90s [Translator's Note: What unit is this]) and bradycardia over 24 hours
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Imaging showed multiple cerebral stenosis (Figure 1), diffusion suppression, and T2/FLAIR hyperintensity (Figure 2); transcranial Doppler (TCD) showed increased blood flow velocity
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A diagnosis of reversible cerebral vasoconstriction syndrome (RCVS) was made
Figure 1.
(A) Spinal hardware implanted after a traumatic accident
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Anterior cervical spine fusion from C4 to C7, and posterior spinal fusion from C4 to upper thoracic spine
Figure 2.
(A) and (B) MRI on admission; DWI (A) and ADC (B) sequences showing acute left parieto-occipital infarction
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(C) and (D) Completed at the time of acute exacerbation of headache and vision loss, DWI (C) and ADC (D) sequences show new ischemic infarction:
Neurogenic bladder can trigger autonomic dysreflexia (ered autonomic dysreflexia, AD), thereby triggering RCV
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Injury above the level of the T6 spinal cord eliminates supraspinal modulation and can lead to AD, an excessive sympathetic reflex, resulting in episodic hypertension and bradycardia.
tachycardia)
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After suprapubic catheter placement, the seizures stopped, the TCD speed was normal, and vision was restored
Injury above the level of the T6 spinal cord eliminates supraspinal modulation and can lead to AD, an excessive sympathetic reflex, resulting in episodic hypertension and bradycardia.
Original source:
Christina M.
Teaching NeuroImage: Neurovascular Consequences of Autonomic Dysreflexia Leave a Comment here