echemi logo
Product
  • Product
  • Supplier
  • Inquiry
    Home > Food News > Food Articles > The resistance mechanism of citrus ulcer disease is revealed.

    The resistance mechanism of citrus ulcer disease is revealed.

    • Last Update: 2020-09-10
    • Source: Internet
    • Author: User
    Search more information of high quality chemicals, good prices and reliable suppliers, visit www.echemi.com
    Original title: Citrus ulcer disease resistance mechanism revealed
    CsWAKL08 is located in the mass membrane
    city citrus
    for the map
    Recently,
    Southwest University
    /
    China
    Institute of Citrus Research of the Agricultural Academy of Sciences researcher Chen Shanchun research group published in Horticultural Research how CsWAKL08 how to improve the resistance of citrus to ulcer disease mechanism.
    , author of the paper, citrus ulcer disease seriously affects the healthy development of the world citrus industry. The exploration of disease-related genes and the analysis of their mechanism of action can provide important genetic resources and theoretical basis for molecular breeding of citrus anti-ulcer disease.
    , which is associated with the cell walls of plants, plays an important role in the response of plants to pathogen invasion. When pathogens are infected, the enzyme can recognize changes in cell wall composition and transmit the intrusion signal through the transmeral domain to the phosphate kinase domain within the cell, regulating downstream disease-resistant genes or signaling pathrapses through the phosphatization of the target protein, triggering plant disease resistance. However, there have been no previous reports in citrus studies that WAKL has been used to increase citrus resistance to ulcer disease.
    the study obtained csWAKL08, a member induced by differences in ulcer bacteria in resistant, sensing varieties, by analyzing excavation and ulcer-induced expression patterns in the citrus WAKL family. CsWAKL08 hyper-expression can significantly enhance citrus resistance to ulcer disease, while gene silencing makes genetically modified plants more sensitive to ulcer disease.
    study found that CsWAKL08 rebuilt reactive oxygen (ROS) balance in hyper-expression plants, increased jasmine acid (JA) accumulation, and increased the expression of PR proteins associated with jasmine acid pathways, thereby increasing citrus resistance to ulcer disease.
    Chen Shanchun said that this is the first time to reveal the mechanism by which plant cell walls are associated with the gene kinase regulation of citrus ulcer disease, CsWAKL08 is a potential disease-resistant gene in the molecular breeding of citrus anti-ulcer disease.
    related paper information:

    .
    This article is an English version of an article which is originally in the Chinese language on echemi.com and is provided for information purposes only. This website makes no representation or warranty of any kind, either expressed or implied, as to the accuracy, completeness ownership or reliability of the article or any translations thereof. If you have any concerns or complaints relating to the article, please send an email, providing a detailed description of the concern or complaint, to service@echemi.com. A staff member will contact you within 5 working days. Once verified, infringing content will be removed immediately.

    Contact Us

    The source of this page with content of products and services is from Internet, which doesn't represent ECHEMI's opinion. If you have any queries, please write to service@echemi.com. It will be replied within 5 days.

    Moreover, if you find any instances of plagiarism from the page, please send email to service@echemi.com with relevant evidence.