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Source: The Third Affiliated Hospital of Guangzhou Medical University Introduction: When hyperuricemia is mentioned, many people will think of gout, and often think that it is more common in middle-aged and elderly m.
As everyone knows, in recent years, more and more patients with asymptomatic hyperuricemia, hyperuricemia has become the fourth "high" after hypertension, hyperglycemia and hyperlipidem.
Does being asymptomatic mean no intervention? Today, the top three use a case to tell everyo.
.
Lei, 39 years old, found that uric acid was as high as 456umol/L in the physical examination, and her basal metabolic age was 54 years o.
Through dietary guidance and health intervention, .
Lei's hyperuricemia not only decreased, but her weight also decreased by 17kg, and the "bucket waist" unexpectedly disappeared! Reminder: Hyperuricemia is not exclusive to middle-aged and elderly m.
Hyperuricemia not only brings about gout, but also induces or aggravates the risk of gouty nephropathy, acute renal failure, hypertension, hyperlipidemia, diabetes and cardiovascular disea.
Risk; Asymptomatic hyperuricemia should be paid more attention, and early scientific diet and exercise can reverse hyperuricem.
At the age of 0139, she was found to have high uric acid! Not only did the uric acid drop after the intervention, but she also lost 34 poun.
.
Lei, 39, is an asymptomatic patient with hyperuricem.
Irregular diet and often staying up la.
Unhealthy living habits not only gave her a "bucket waist", but also detected uric acid as high as 456umol/L in a routine physical examination, and her basal metabolic age reached 54 years old! Uric acid is the product of purine metabolism in the human body, and it itself has an antioxidant effe.
The disorder of purine metabolism in the body leads to hyperuricem.
Clinically, hyperuricemia (HUA) means that the fasting blood uric acid level is higher than 420 μmol/L in men and 360 μmol/L in women on two different days, which is hyperuricem.
Having never had gout symptoms, .
Lei was surprised that she developed hyperuricem.
Aware of the potential harm of high uric acid to the body, I went to the hospital to see a doct.
Under my guidance, I started the road of health interventi.
After carefully studying .
Lei's body data, I developed a personalized diet nutrition prescription and weight management plan for h.
After more than two months of health intervention, her uric acid value finally returned to a normal level of 289umol/L, and her weight dropped from 76kg to 57.
Both weight loss and uric acid reduction achieved initial resul.
△ Before and after the health intervention, the comparison of the patient's body shape △ Before and after the health intervention, the comparison of the patient's health data 02 Exclusively for men? Just gout? Four common misunderstandings of hyperuricemia Hyperuricemia, a disease that used to belong only to middle-aged and elderly people, seems to be getting younger and young.
High uric acid is easily associated with diseases such as hypertension, abnormal lipid metabolism, diabetes, and obesi.
Long-term high-calorie diet, lack of exercise, and heavy drinking can easily lead to these proble.
There are four common misconceptions about hyperuricemia: Misunderstanding 1: Only middle-aged men suffer from hyperuricemia? Although now 95% of gout patients are male, and the high incidence age group is over 30 years o.
But hyperuricemia is not just for middle-aged m.
On the one hand, living standards have improved, and the purine content in the diet is getting higher and higher, which makes many young people who do not pay attention to their diet also suffer from hyperuricem.
On the other hand, because women after menopause have lower levels of estrogen that can promote uric acid excretion, they cannot be careless about go.
Myth 2: Hyperuricemia must have symptoms? No need for treatment without symptoms? Many people have a high uric acid level in the physical examination, but they have never experienced gout symptoms and feel that they have no pain and do not need treatme.
This idea is wro.
Not all hyperuricemias will have symptoms, and only about one-third of hyperuricemia patients will develop go.
Asymptomatic hyperuricemia refers to patients with only hyperuricemia without clinical symptoms such as arthritis, tophi, and uric acid ston.
However, regardless of whether a person has a symptom attack or not, as long as the fasting blood uric acid level detected on different two days exceeds the normal value, it is hyperuricemia, and treatment measures should be taken in ti.
If the patient also has diabetes, chronic kidney disease or risk factors for cardiovascular disease, he should choose a scientific treatment method immediately according to his physical conditi.
Myth 3: Does hyperuricemia only cause gout? High blood uric acid is the main cause of go.
When the concentration of uric acid in the blood reaches a saturated state, it will precipitate out of the blood and produce urate crysta.
These crystals are deposited in joints such as ankles and toes, causing local inflammation, showing redness, swelling, heat, pain and dysfuncti.
This symptom is go.
However, hyperuricemia does not only bring about gout, long-term high uric acid can also cause kidney, cardiovascular, pancreatic β-cell damage, induce or aggravate gouty nephropathy, acute renal failure, hypertension, hyperlipidemia, Risk of diabetes and cardiovascular disea.
Myth 4: After the blood uric acid index is normal, there will be no gout? A normal blood uric acid value does not mean that the uric acid level in the human body is norm.
Many gout patients only pay attention to the blood uric acid level and ignore the key step in the treatment of gout - dissolving ura.
As long as there are urate crystals in the body, gout will recur aga.
The latest guidelines recommend that the uric acid level of gout patients should be reduced to below 360 μmol/L, and those with renal uric acid stones should be reduced to below 300 μmol/L, and the formed tophi should be reduced to further avoid the deposition of urate crystals in the bo.
To reduce the frequency of gout recurren.
03 Do these three points well! Prevention and early intervention of hyperuricemia There are two main sources of uric acid in the human body: one is food intake, which only accounts for 10%-20% of the total uric ac.
The second is its own purine metabolism, accounting for about 8
Uric acid is mainly excreted through the kidneys and intestines, and the kidneys are the main excretion method, accounting for about 8
The prevention and intervention of hyperuricemia mainly lies in adjusting the life>
Balanced diet, eat less high-purine foods, and eat more low-purine foods, such as cereals, nuts, eggs, low-fat dairy products, vegetables, fruits,e.
Eat moderate amounts of purine foods, such as meat, aquatic products, and soy produc.
High-purine foods should be eaten less, such as animal offal, shellfish, and sou.
Pay attention to a balanced diet, try to achieve low salt, low sugar, low oil, low calorie! Drink more water, drink less beverages and alcoh.
The daily drinking water should be no less than 5L-2L, which is equivalent to 8~10 glasses of wat.
Water intake should be mainly boiled water, fresh tea, mineral water,e.
Avoid high sugar intake such as carbonated beverages, fructose-containing beverages,e.
Drink less alcohol, generally recommend not drinking beer and liqu.
Alcohol in wine can lead to the accumulation of lactic acid and ketones in the bo.
These two substances will compete with uric acid for excretion channels during urination, and eventually reduce the excretion of uric acid, thereby causing hyperuricem.
More exercise, regular work and re.
Regular work and rest habits have a very positive effect on enhancing one's own immunity and improving physical fitne.
Patients with hyperuricemia can do aerobic exercise, such as jogging, walking, tai chi,e.
Be careful to avoid intense anaerobic exercise, such as sprinting, high-speed swimming, and strength traini.
Because anaerobic exercise will decompose adenosine triphosphate (ATP) in large quantities to produce adenine, which will increase the level of uric acid and aggravate go.
The harm of hyperuricemia is potential and persistent, and the long-term harm is even greater, which must be paid attention to! Be vigilant, develop good living habits, and monitor uric acid regular.
To achieve early detection, early intervention, and early treatment, so as not to drag small problems into big proble.
Don't talk about "high" anymore, let's act quickly and refuse high uric acid! (The attending physician and medical guidance of this case: Zeng Qingshan, Deputy Chief Physician of the Department of Clinical Nutrition, The Third Affiliated Hospital of Guangzhou Medical University; Source: The Third Affiliated Hospital of Guangzhou Medical University) If you can see this article, please forward it to the circle of friends to let More people know a little more! Stars follow the wonderful stories of "The Last Dopamine" WeChat official account Never miss .
I used a stethoscope to measure the lives of people who met by chance, and those lives that passed by in my hands are piled up in the corners of my eyes drop by dr.
Tears explained to me the meaning of li.
I am the last dopamine, recording sounds from the real wor.
Some pictures are from the Intern.
If there is any infringement, please contact the background in ti.
Copyright, cooperation WeChat: dba0604 Submission email: last-dopamine@foxma.
com
As everyone knows, in recent years, more and more patients with asymptomatic hyperuricemia, hyperuricemia has become the fourth "high" after hypertension, hyperglycemia and hyperlipidem.
Does being asymptomatic mean no intervention? Today, the top three use a case to tell everyo.
.
Lei, 39 years old, found that uric acid was as high as 456umol/L in the physical examination, and her basal metabolic age was 54 years o.
Through dietary guidance and health intervention, .
Lei's hyperuricemia not only decreased, but her weight also decreased by 17kg, and the "bucket waist" unexpectedly disappeared! Reminder: Hyperuricemia is not exclusive to middle-aged and elderly m.
Hyperuricemia not only brings about gout, but also induces or aggravates the risk of gouty nephropathy, acute renal failure, hypertension, hyperlipidemia, diabetes and cardiovascular disea.
Risk; Asymptomatic hyperuricemia should be paid more attention, and early scientific diet and exercise can reverse hyperuricem.
At the age of 0139, she was found to have high uric acid! Not only did the uric acid drop after the intervention, but she also lost 34 poun.
.
Lei, 39, is an asymptomatic patient with hyperuricem.
Irregular diet and often staying up la.
Unhealthy living habits not only gave her a "bucket waist", but also detected uric acid as high as 456umol/L in a routine physical examination, and her basal metabolic age reached 54 years old! Uric acid is the product of purine metabolism in the human body, and it itself has an antioxidant effe.
The disorder of purine metabolism in the body leads to hyperuricem.
Clinically, hyperuricemia (HUA) means that the fasting blood uric acid level is higher than 420 μmol/L in men and 360 μmol/L in women on two different days, which is hyperuricem.
Having never had gout symptoms, .
Lei was surprised that she developed hyperuricem.
Aware of the potential harm of high uric acid to the body, I went to the hospital to see a doct.
Under my guidance, I started the road of health interventi.
After carefully studying .
Lei's body data, I developed a personalized diet nutrition prescription and weight management plan for h.
After more than two months of health intervention, her uric acid value finally returned to a normal level of 289umol/L, and her weight dropped from 76kg to 57.
Both weight loss and uric acid reduction achieved initial resul.
△ Before and after the health intervention, the comparison of the patient's body shape △ Before and after the health intervention, the comparison of the patient's health data 02 Exclusively for men? Just gout? Four common misunderstandings of hyperuricemia Hyperuricemia, a disease that used to belong only to middle-aged and elderly people, seems to be getting younger and young.
High uric acid is easily associated with diseases such as hypertension, abnormal lipid metabolism, diabetes, and obesi.
Long-term high-calorie diet, lack of exercise, and heavy drinking can easily lead to these proble.
There are four common misconceptions about hyperuricemia: Misunderstanding 1: Only middle-aged men suffer from hyperuricemia? Although now 95% of gout patients are male, and the high incidence age group is over 30 years o.
But hyperuricemia is not just for middle-aged m.
On the one hand, living standards have improved, and the purine content in the diet is getting higher and higher, which makes many young people who do not pay attention to their diet also suffer from hyperuricem.
On the other hand, because women after menopause have lower levels of estrogen that can promote uric acid excretion, they cannot be careless about go.
Myth 2: Hyperuricemia must have symptoms? No need for treatment without symptoms? Many people have a high uric acid level in the physical examination, but they have never experienced gout symptoms and feel that they have no pain and do not need treatme.
This idea is wro.
Not all hyperuricemias will have symptoms, and only about one-third of hyperuricemia patients will develop go.
Asymptomatic hyperuricemia refers to patients with only hyperuricemia without clinical symptoms such as arthritis, tophi, and uric acid ston.
However, regardless of whether a person has a symptom attack or not, as long as the fasting blood uric acid level detected on different two days exceeds the normal value, it is hyperuricemia, and treatment measures should be taken in ti.
If the patient also has diabetes, chronic kidney disease or risk factors for cardiovascular disease, he should choose a scientific treatment method immediately according to his physical conditi.
Myth 3: Does hyperuricemia only cause gout? High blood uric acid is the main cause of go.
When the concentration of uric acid in the blood reaches a saturated state, it will precipitate out of the blood and produce urate crysta.
These crystals are deposited in joints such as ankles and toes, causing local inflammation, showing redness, swelling, heat, pain and dysfuncti.
This symptom is go.
However, hyperuricemia does not only bring about gout, long-term high uric acid can also cause kidney, cardiovascular, pancreatic β-cell damage, induce or aggravate gouty nephropathy, acute renal failure, hypertension, hyperlipidemia, Risk of diabetes and cardiovascular disea.
Myth 4: After the blood uric acid index is normal, there will be no gout? A normal blood uric acid value does not mean that the uric acid level in the human body is norm.
Many gout patients only pay attention to the blood uric acid level and ignore the key step in the treatment of gout - dissolving ura.
As long as there are urate crystals in the body, gout will recur aga.
The latest guidelines recommend that the uric acid level of gout patients should be reduced to below 360 μmol/L, and those with renal uric acid stones should be reduced to below 300 μmol/L, and the formed tophi should be reduced to further avoid the deposition of urate crystals in the bo.
To reduce the frequency of gout recurren.
03 Do these three points well! Prevention and early intervention of hyperuricemia There are two main sources of uric acid in the human body: one is food intake, which only accounts for 10%-20% of the total uric ac.
The second is its own purine metabolism, accounting for about 8
Uric acid is mainly excreted through the kidneys and intestines, and the kidneys are the main excretion method, accounting for about 8
The prevention and intervention of hyperuricemia mainly lies in adjusting the life>
Balanced diet, eat less high-purine foods, and eat more low-purine foods, such as cereals, nuts, eggs, low-fat dairy products, vegetables, fruits,e.
Eat moderate amounts of purine foods, such as meat, aquatic products, and soy produc.
High-purine foods should be eaten less, such as animal offal, shellfish, and sou.
Pay attention to a balanced diet, try to achieve low salt, low sugar, low oil, low calorie! Drink more water, drink less beverages and alcoh.
The daily drinking water should be no less than 5L-2L, which is equivalent to 8~10 glasses of wat.
Water intake should be mainly boiled water, fresh tea, mineral water,e.
Avoid high sugar intake such as carbonated beverages, fructose-containing beverages,e.
Drink less alcohol, generally recommend not drinking beer and liqu.
Alcohol in wine can lead to the accumulation of lactic acid and ketones in the bo.
These two substances will compete with uric acid for excretion channels during urination, and eventually reduce the excretion of uric acid, thereby causing hyperuricem.
More exercise, regular work and re.
Regular work and rest habits have a very positive effect on enhancing one's own immunity and improving physical fitne.
Patients with hyperuricemia can do aerobic exercise, such as jogging, walking, tai chi,e.
Be careful to avoid intense anaerobic exercise, such as sprinting, high-speed swimming, and strength traini.
Because anaerobic exercise will decompose adenosine triphosphate (ATP) in large quantities to produce adenine, which will increase the level of uric acid and aggravate go.
The harm of hyperuricemia is potential and persistent, and the long-term harm is even greater, which must be paid attention to! Be vigilant, develop good living habits, and monitor uric acid regular.
To achieve early detection, early intervention, and early treatment, so as not to drag small problems into big proble.
Don't talk about "high" anymore, let's act quickly and refuse high uric acid! (The attending physician and medical guidance of this case: Zeng Qingshan, Deputy Chief Physician of the Department of Clinical Nutrition, The Third Affiliated Hospital of Guangzhou Medical University; Source: The Third Affiliated Hospital of Guangzhou Medical University) If you can see this article, please forward it to the circle of friends to let More people know a little more! Stars follow the wonderful stories of "The Last Dopamine" WeChat official account Never miss .
I used a stethoscope to measure the lives of people who met by chance, and those lives that passed by in my hands are piled up in the corners of my eyes drop by dr.
Tears explained to me the meaning of li.
I am the last dopamine, recording sounds from the real wor.
Some pictures are from the Intern.
If there is any infringement, please contact the background in ti.
Copyright, cooperation WeChat: dba0604 Submission email: last-dopamine@foxma.
com
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