Flow: Immune thrombosis in neocooproid pneumonia is associated with respiratory failure and systemic high coagulation.

The new crown pneumonia virus ravaged the world, from time to time, autumn and winter is about to, to prevent the outbreak of wildfires to rekindle the task of a second outbreak is becoming more and more urgent.
The virus infection not only causes severe pneumonia (COVID-19) and respiratory failure, but also accumulates the kidneys and heart muscle, but so far, the mechanisms leading to respiratory failure and heart and kidney injury have not been fully defined.
wave of uneven, wave after wave, COVID-19 patients with systemic thrombosis esopolysis is also common reports.
study included 62 individuals, including 38 COVID-19 patients diagnosed with qPCR and 24 non-COVID-19 controls.
researchers conducted an histological pathological assessment of autopsy cases, a ideotype analysis of neutral granulocytes and plateplates based on surface markers, and a determination of plateplate, neutral granulocyte function and clotting function.
(COVID-19-related microvascular thrombosis in the lungs, kidneys and heart) showed that organ injury and thrombosis characteristics in PATIENTs with COVID-19 were associated with immunothrombosis.
in patients with COVID-19, inflammatory microvascular thrombosis is present in the lungs, kidneys and heart and contains neutrinophilic extracellular traps associated with plate plateboard and fibrin.
(neutral granulocyte activation in patients with severe COVID-19) COVID-19 patients also exhibited neutral granulocyte-plate plateplate aggregation, as well as significant patterns of neutral granulocyte and plate platea activation in the blood, which vary with the severity of the disease.
Compared to healthy controlled and non-COVID-19 pneumonia individuals, patients with moderate COVID-19 pneumonia showed plate plate plateboard depletion and low activation esoteric esotericity of neutral granulocytes, while severe patients showed excessive activation of plate plate plates and neutral granulocytes.
abnormal immune thrombosis in patients with severe neo-coronary pneumonia were associated with acute respiratory distress syndrome and systemic high coagulation.
, the study shows that immunothrombosis is a key marker of the severity of COVID-19 disease.
further research is needed to determine the role of immunothrombosis in COVID-19.
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