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With obesity and obesity-related diseases (such as non-alcoholic fatty liver NAFLD, type 2 diabetes mellitus T2DM and medsci.
Non-alcoholic fatty liver disease diabetes medsci.
Even eating sugary drinks with a moderate amount of fructose within a few weeks can cause changes in the composition of serum fatty acids, leading to liver insulin resistance.
The specific effect of fructose stems from the liver's major role in fructose clearance.
The specific effect of fructose stems from the liver's major role in fructose clearance.
The subject’s metabolism has undergone significant changes, resulting in a doubling of the human liver’s fat production, and the subject’s metabolism has undergone significant changes, resulting in a doubling of the human liver’s fat production.
A total of 94 subjects were included in this double-blind randomized trial.
In general, the participants did not consume more calories than before the study.
Participants who consumed free fructose, fructose and glucose (sucrose) beverages daily increased the basal secretion (FSR) of newly synthesized fatty acids in the liver by 2 times (median FSR %/day in the SSB group: sucrose = 20.
In addition, fructose intake does not change the basal secretion of newly synthesized VLDL-TAG.
It can be seen that regular consumption of medium-dose fructose and sucrose-sweetened beverages is associated with a stable calorie intake, even in the basal state, it will increase liver fatty acid synthesis, but this effect is not observed after consuming glucose.
Frequent consumption of medium-dose fructose and sucrose-sweetened beverages is associated with a stable calorie intake, even in the basal state, it will increase liver fatty acid synthesis, but this effect was not observed after consuming glucose.
Although soft drinks are delicious, long-term intake of sugary drinks will accelerate the development of diabetes or fatty liver.
references:
Fructose- and sucrose- but not glucose-sweetened beverages promote hepatic de novo lipogenesis: A randomized controlled trial.
Fructose- and sucrose- but not glucose-sweetened beverages promote hepatic de novo lipogenesis: A randomized controlled trial.
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