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Nat Commun: Improving the diversity of intestinal flora in patients with acute myeloid leukemia after autologous fecal bacteria transplantation

 Last Update: 2021-06-01
 Source: Internet
 Author: User

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Acute myeloid leukemia (AML) is a rare but potentially fatal blood cancer.

Previous studies have shown that the high diversity of intestinal microbes is closely related to the improvement of clinical outcomes of AML patients and the reduction of infection complications.

Infection antibiotics

In this study, the researchers reported the results of a phase II single-arm, multi-center study, which mainly evaluated data related to autologous fecal microbiota transplantation (AFMT) in 25 AML patients who received intensive chemotherapy and antibiotic therapy.

The researchers reported on the results of a phase II single-arm, multi-center study, which mainly evaluated data related to autologous fecal microbiota transplantation (AFMT) in 25 AML patients who received intensive chemotherapy and antibiotic therapy.

Research flow chart

The main result of this study is to evaluate the efficacy of AFMT in correcting homeostasis disorders and multi-drug resistant bacteria .

The main result of this study was to evaluate AFMT in correcting homeostasis and multiple effects resistant bacteria aspects of the main results of this study was to evaluate the effectiveness of corrective AFMT in homeostasis and multidrug-resistant bacteria aspects of immunity

Fecal microbiota characteristics of patients in each regimen

The results show that intensive induction chemotherapy can lead to a sharp decline in the alpha-diversity index of the microbial community, accompanied by significant changes in the microbial community and microbial malnutrition caused by the regulation of pro-inflammatory families.

Intensified induction chemotherapy can lead to a sharp decline in the alpha-diversity index of the microbial community, accompanied by significant changes in the microbial community and microbial malnutrition caused by the regulation of pro-inflammatory families.

org/10.

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